Abstract
Strains derived from HfrH carrying the arcA2 null mutation exhibit a higher respiratory rate, enhanced glucose consumption, and a more-reduced intracellular redox state than arcA deletion mutants of a different lineage. The phenotype of the arcA2 mutants was due to the presence of a creC constitutive mutation introduced by PI transduction.
Original language | English |
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Journal | Journal of Bacteriology |
Volume | 190 |
Issue number | 9 |
Pages (from-to) | 3404-3407 |
Number of pages | 4 |
ISSN | 0021-9193 |
DOIs | |
Publication status | Published - 2008 |
Externally published | Yes |