Antioxidant properties of modified rutin esters by DPPH, reducing power, iron chelation and human low density lipoprotein assays

Bena-Marie Lue, Nina Skall Nielsen, Charlotte Jacobsen, Lars Hellgren, Zheng Guo, Xuebing Xu

Research output: Contribution to journalJournal articleResearchpeer-review

Abstract

Practical limitations exist regarding the effectiveness of flavonoids as antioxidants in many food systems, possibly due to their poor solubility and miscibility in lipidic environments. Current strategies to improve these properties include enzymatically acylating flavonoids with lipophilic moieties. Herein, two derivatives of rutin (possessing C12:0 or C16:0 acyl groups) were assessed for their antioxidant properties, and compared with their parent compound, rutin and with butylated hydroxytoluene (BHT). While all compounds exhibited relatively strong radical scavenging abilities, modified rutin compounds exhibited decreased reducing power and metal chelating abilities as compared to rutin. Conversely, investigations on the oxidation of human low density lipoprotein (LDL) revealed that rutin laurate was most effective in inhibiting oxidation by prolonging LDL lag time for an in vitro system. With regards to in vivo considerations, a pre-treatment step confirmed that the ester bond linking rutin and acyl moieties was most susceptible to hydrolysis by digestive enzymes, while rutin itself was not degraded. Thus, acylation of rutin with medium or long chain fatty acids may result in improved antioxidant abilities in more complex systems, including LDL-oxidation assays. Likely reasons may include improved lipophilic solubility and partitioning properties allowing for better accessibility to the actual site of oxidation. (C) 2010 Elsevier Ltd. All rights reserved.
Original languageEnglish
JournalFood Chemistry
Volume123
Issue number2
Pages (from-to)221-230
ISSN0308-8146
DOIs
Publication statusPublished - 2010

Fingerprint

Dive into the research topics of 'Antioxidant properties of modified rutin esters by DPPH, reducing power, iron chelation and human low density lipoprotein assays'. Together they form a unique fingerprint.

Cite this