Alexandr Parlesak

Alexandr Parlesak


Background of research:

Many diseases of affluence would not develop without immune dysregulation: cardiovascular disease, allergies, asthma, and others. A key trigger of inflammatory events in the body is the intestinal flora. If microbial structures from the gut lumen manage to cross the intestinal barrier, sub-clinical chronic inflammation may occur, promoting disorders mentioned above.
Nutrition plays an important role in maintaining the intestinal barrier and keeping the delicate balance between tolerance to harmless structures and a fast immune response against pathogens.

Focus of research:

- Identification of key structures from the
  intestine triggering sub-clinical chronic
- Investigation of food components counteracting
  activation of immune cells
- Development of nutritional strategies for an
  enhanced intestinal integrity
- Establishment of models depicting the systemic
  interaction among mucosal cells, immune cells,
  intestinal microbiota, and nutrients

Goal of research:

To find a systemic approach for effective prevention of nutrition-related immune disorders

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  • J D D G (Print Edition)

    ISSNs: 1610-0379

    Additional searchable ISSN (Electronic): 1610-0387

    Wiley-Blackwell Verlag GmbH, Germany

    BFI (2017): BFI-level 1, Scopus rating (2016): CiteScore 1.15 SJR 0.56 SNIP 1.302, ISI indexed (2013): ISI indexed yes, Web of Science (2017): Indexed Yes

    Central database


  • Journal of Nutrition

    ISSNs: 0022-3166

    Additional searchable ISSN (Electronic): 1541-6100

    American Society for Nutrition, United States

    BFI (2017): BFI-level 2, Scopus rating (2016): CiteScore 3.93 SJR 1.956 SNIP 1.335, ISI indexed (2013): ISI indexed yes, Web of Science (2017): Indexed yes

    Central database


  • British Journal of Nutrition

    ISSNs: 0007-1145, 1475-2662

    Additional searchable ISSN (Electronic): 0954-4224

    Cambridge University Press, United Kingdom

    BFI (2017): BFI-level 1, Scopus rating (2016): CiteScore 3.46 SJR 1.983 SNIP 1.533, ISI indexed (2013): ISI indexed yes, Web of Science (2017): Indexed yes

    Central database


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ID: 200086